Given the spread of misinformation about COVID-19, Just Facts is providing a trove of rigorously documented facts about this disease and its impacts. These include some essential facts that have been absent or misreported in much of the media’s coverage of this issue.
This research also includes a groundbreaking study to determine the lethality of COVID-19 based on the most comprehensive available measure: the total years of life that it will rob from all people. This accords with the Centers for Disease Control’s tenet that “the allocation of health resources must consider not only the number of deaths by cause” but also the “years of potential life lost.”
On one hand, these facts show that:
- The average death rate for people who contract COVID-19 is well below 1% and is much closer to that of the seasonal flu than figures that were commonly reported by the press.
- The average years of life lost from each COVID-19 death are significantly fewer than from common causes of untimely death like accidents and suicides.
- The virus that causes COVID-19 is “very vulnerable to antibody neutralization” and has very limited ability to mutate, which makes it unlikely to take masses of lives year after year like the flu and other recurring scourges.
- If 500,000 COVID-19 deaths ultimately occur in the United States – or more than twice the level of a prominent projection – the disease will rob about 6.8 million years of life from all Americans who were alive at the outset of 2020. In contrast:
- the flu will rob them of about 35 million years.
- suicides will rob them of 132 million years.
- accidents will rob them of 409 million years.
On the other hand, elderly people and those with chronic ailments are extremely vulnerable to COVID-19, and the disease is highly transmissible, which means it could spread like wildfire and overwhelm hospitals without extraordinary measures to contain it. This would greatly increase its death toll.
However, measures to limit the spread of C-19 sometimes involve fatal tradeoffs, and thus, overreactions can destroy more years of life than they save.
Just Facts asked Dr. Rodney X. Sturdivant – who has a doctorate in biostatistics and is the director of the Statistical Consulting Center at Baylor University and an associate professor of statistics – to critically review this research. He assessed it as follows:
This research clearly articulates what is known about COVID-19 and accurately reflects the true risks and ramifications that are critical for policy and individual decisions. The facts in this article reflect analyses that led me to co-sign – along with over 50,000 medical and public health scientists and practitioners – the Great Barrington Declaration. This calls for a policy of ‘focused protection’ to ‘minimize mortality and social harm until we reach herd immunity.’
Likelihood Of Exposure
According to the U.S. Centers for Disease Control and Prevention, a total of 15,271,571 cases of COVID-19 have been reported in the U.S. as of Dec. 9. Given that the population is 331 million, this amounts to one out of every 22 people in the U.S. C-19 cases are not equally dispersed throughout the nation, so this figure is much higher in some areas and much lower in others.
Nevertheless, the current likelihood of being exposed to C-19 doesn’t depend on the number of people who have ever been infected – but on the number of people who are currently infected and contagious. Determining this is confounded by three key factors:
- Symptoms of C-19 typically don’t appear until two to 14 days after exposure, which means that people can be infected for days or weeks before having any reason to get tested.
- The vast majority of people who contract C-19 develop only mild symptoms or none at all, and hence, they may never be diagnosed while they are infectious. Despite widespread anxiety over C-19, medical studies have been clear from early on that it poses such a minor threat to most people that they may barely know they have it:
- A February study in the Journal of the American Medical Association based on data from China found that 81% of reported C-19 cases are “mild” and that the true figure is even higher because there are “inherent difficulties in identifying and counting mild and asymptomatic cases.”
- In one of the few cases where asymptomatic cases could be reliably counted because an entire population was tested for C-19, Japan’s National Institute for Infectious Diseases reported in February that 51% of the people on the Diamond Princess cruise ship who tested positive had no symptoms, even though the “passengers onboard were generally of an older age.” The portion of these people who later developed symptoms is currently unavailable.
- A March paper in the journal Microbes and Infection notes that “most infected individuals … appear to be able to recover with little to no medical intervention.”
- In another case where asymptomatic cases were reliably counted, the New England Journal of Medicine reported in April that universal COVID-19 testing of pregnant women at two New York City hospitals found that 88% of them who tested positive for the disease were asymptomatic upon admission, and 9% later developed a fever. Fevers are relatively common after childbirth, which means they were not necessarily from C-19. Accounting for both possibilities and the sampling margin of error, these results amount to a 65% to 99% asymptomatic rate with 95% confidence.
- Perhaps most tellingly, the journal BMJ Thorax published a study in May about an outbreak of C-19 during an Antarctic cruise where everyone onboard “had no outside human contact” for 28 days. The passengers were quarantined in their rooms on the eighth day of the cruise, the crew wore personal protective equipment thereafter, and everyone was tested for C-19 on day 20. Among the 128 people who tested positive, 81% ultimately had no symptoms, 13% had mild symptoms, and 6% had serious symptoms, including one that died.
- There are significant differences in the contagiousness of people infected with C-19, depending on whether they are symptomatic, pre-symptomatic, post-symptomatic, or asymptomatic. This reality has been shrouded in many publications because researchers often misuse an indicator called “viral load” to determine if people can transmit the virus.
In the context of COVID-19, viral load is the concentration of the virus’ genetic material detected in body fluids. The Journal of Clinical Virology notes that this measure has been “widely used” in C-19 studies “as a proxy for contagiousness,” but a German medical journal explains that the mere detection of genetic material “does not imply presence of viable and replicating virus.”
In other words, the virus may be dead or unable to reproduce because the person’s immune system has neutered it. The German journal adds that researchers commonly use viral load to measure contagiousness because it is “convenient,” while the proper method is “cumbersome.”
The implications of using a convenient measure instead of an accurate one are evidenced by the fact that an August paper in JAMA Internal Medicine found that C-19 viral loads “were similar” in people during different stages of the disease – but studies that actually measure contagiousness show major disparities in their capacity to transmit infections:
- An April paper in the journal Nature reports that live C-19 viruses could be “readily” cultured from the mucus of people with “mild” C-19 “during the first week of symptoms” but not “after day eight in spite of ongoing high viral loads.”
- An April paper in the journal Nature Medicine estimates that 1% of C-19 transmission occurs more than five days before symptom onset and that infectiousness peaks at symptom onset and then declines “quickly within seven days.”
- An August contact tracing study in the journal Respiratory Medicine found that none of 455 people who were exposed to an “asymptomatic COVID-19 virus carrier” caught the disease, even though each of them were exposed for an average of four to five days. The paper thus concludes that “the infectivity of some asymptomatic” C-19 virus “carriers might be weak.”
- A contact tracing study published by the Annals of Internal Medicine in August found that merely one among 342 people who had “close contact” with asymptomatic C-19 virus carriers caught the disease from them. Based on this and other data, the study determined that the contagiousness of asymptomatic C-19 carriers is 91% lower than those with mild symptoms and 95% lower than those with moderate symptoms.
Applying the facts above and related data from the CDC and the Center for Evidence-Based Medicine at Oxford University, Just Facts developed a methodology to estimate the number of people in the U.S. who currently have infectious cases of COVID-19. The calculations – which are detailed in this spreadsheet – find that roughly one of every 54 people in the U.S. was contagious as of Dec. 9.
Again, this is a national average and is not applicable everywhere because of large differences in the timing and severity of the pandemic in different areas of the country.
Fatal interactions between COVID-19 and other ailments like heart disease and diabetes make it difficult to determine the exact death toll from C-19. A range of evidence suggests that the CDC’s tally of COVID-19 deaths may be modestly inflated but still provides a reasonable measure of the pandemic’s severity.
According to the CDC’s estimate of “confirmed and probable” fatalities from COVID-19, a total of 288,762 U.S. residents have died from the disease as of Dec. 9. To put this figure in perspective:
- C-19 has killed about one out of every 1,145 Americans, whereas one out of every 116 Americans die every year.
- an average of 37,000 people per year died from the flu over the past nine years.
- around 170,000 people die from accidents every year.
In comparative terms, deaths from COVID-19 are currently about:
- 90% below annual fatalities from all causes.
- 7.7 times average annual fatalities from the flu.
- 70% above annual fatalities from accidents.
Although C-19 is a new disease and took its first reported life in the U.S. during late February, these comparisons may substantially overstate its deadliness. This is because fatalities from accidents, the flu, and other common causes of death occur in droves every year, and this is unlikely for C-19 because of the facts below.
The primary reason why the flu kills tens of thousands of people every year is that the viruses that cause it mutate in ways that prevent humans from becoming immune to them. Per the Journal of Infectious Diseases, “All viruses mutate, but influenza remains highly unusual among infectious diseases” because it mutates very rapidly, and thus, “new vaccines are needed almost every year” to protect against it.
In striking contrast, a March paper in a molecular biology journal that cites Michael Farzan, co-chair of the Department of Immunology and Microbiology at Scripps Research, explains that once a vaccine for COVID-19 is developed, it “would not need regular updates, unlike seasonal influenza vaccines.” This is because the part of the virus that the vaccine targets “is protected against mutation” by a feature of its genetic material, or RNA.
The same point applies to naturally acquired immunity. People who catch C-19 and other infectious diseases with low rates of mutation typically develop enduring resistance to them. The physiology textbook The Human Body in Health and Illness explains that such immunity, which is called “active immunity,” is “generally long lasting.”
This is the case with rubella, mumps, measles, and polio. After people contract these diseases and recover, they rarely get them again or transmit them to others. Thus, they become firewalls against the spread of these contagions.
Media outlets like The Atlantic, Vox, and Forbes have turned the truth of this matter on its head by confusing the general nature of coronaviruses with that of C-19. The habit of calling COVID-19 “the coronavirus” can be very misleading because there are different types of coronaviruses, and C-19 is caused by just one of them.
Coronaviruses are a family of RNA viruses that include some common cold viruses that tend to mutate rapidly, but the virus that causes C-19 does not share that trait. Per the molecular biology paper cited above, the virus that causes C-19 “does not mutate rapidly for an RNA virus because, unusually for this category, it has a proof-reading function” in its genetics. The paper also explains that the same physical feature of the virus that makes it so contagious also makes it:
Very vulnerable to antibody neutralization, and thus it is a relatively easy virus to protect against. I refer to it as ‘stupid’ on a spectrum where HIV, which lives in the face of an active immune system for years, is a ‘genius.’
Simply put, the virus that causes C-19 is immunologically fragile, and thus, it is far less likely to take lives regardless of acquired immunity and vaccines. If this proves true in the long run – as all available evidence suggests it will – the lifetime risk of dying from COVID-19 is greatly overstated by comparing its current death toll to yearly fatalities from the flu, accidents, suicides, and other common causes of death. This is because the vast bulk of people will develop immunity to C-19 either by catching it or through vaccines. In vivid contrast, societies don’t become immune to accidents, the flu, or heart disease.
All of the above facts about immunology in general and C-19 in particular were published in medical journals, textbooks, or encyclopedias near the outset of the pandemic in March or earlier. Yet, the World Health Organization sowed confusion around this issue by publishing a scientific brief and a tweet on April 24 that stated: “There is currently no evidence that people who have recovered from #COVID19 and have antibodies are protected from a second infection.”
Many media outlets parroted that claim, even though the World Health Organization deleted its tweet later that day and wrote: “Earlier today we tweeted about a new WHO scientific brief on ‘immunity passports’. The thread caused some concern & we would like to clarify: We expect that most people who are infected with #COVID19 will develop an antibody response that will provide some level of protection.”
That clarification was closer to the truth but still misleading because it ignored these three key facts that were already proven by that time:
- The virus that causes C-19 has low mutational capacity.
- The virus that causes C-19 is very vulnerable to antibody neutralization.
- Viruses with these attributes are easy to protect against because vaccines and acquired immunity commonly provide long-lasting immunity against them.
Since April, a torrent of research has continued to confirm those facts:
- In May, the South Korea Centers for Disease Control published a study of 285 people who contracted C-19, recovered, and then tested positive again. Contrary to the assumption that they must have caught the disease a second time, the study found no evidence that even one these individuals was infectious. Viral culture testing was performed on 108 of the subjects, and “all had negative results.” Also, “no case was found” in which someone “was newly infected solely from contact with” one of the people. These facts indicate that these positive C-19 tests were caused by remnants of dead or harmless virus particles. Thus, South Korea changed its policy to treat such individuals as non-infectious.
- In August, the journal Cell published a study of the immune system responses of 206 people in Sweden who had contracted C-19. Unlike other studies that only looked for antibodies, this one measured “T cells,” which are the body’s main source of “long-term immune protection.” This is important because antibodies quickly wane after infections, while T cells provide lasting immunity. The study found that:
- 100% of the subjects who had severe C-19 and 87% who had mild C-19 had “highly functional memory T cell responses, suggesting that natural exposure or infection may prevent recurrent episodes of severe COVID-19.”
- “In line with these observations, none of the convalescent individuals in this study, including those with previous mild disease, have experienced further episodes of COVID-19.”
- In August, after C-19 had “affected over 23 million patients with more than 0.8 million deaths in over 200 countries,” the journal Clinical Infectious Diseases reported the first proven case of someone catching C-19 and then catching it again. This sometimes occurs with all infectious diseases – including with people who have been vaccinated – because immunity is not an all-or-nothing phenomenon. However, when it comes to C-19 and other ailments caused by viruses that are vulnerable to antibodies and have little capacity for mutations, such events are rare exceptions, not the rule.
- In September, the Proceedings of the National Academy of Sciences published a genetic analysis of 18,514 virus samples collected from people who contracted C-19 in 84 different nations. The study found that mutations to date have been so “limited” that “a single vaccine” should be “broadly protective” on a “global” basis. The authors note this situation is very different from the viruses that cause HIV, the flu, and Dengue fever because their ample mutations make it nearly impossible to create effective vaccines against them.
- In November, the journal Cell published a study of 15 people who recovered from mild cases of C-19 to determine if their immune systems developed “a multilayered defense that lasts.” The study found that all of them did, as evidenced by the presence of C-19-specific T cells and B cells in their blood. Both are sources of long-term immune protection, although T cells are the primary source. Researchers also tested the cells and found that they reacted appropriately when exposed to the virus that causes C-19. Those results, as the authors point out, are consistent with the fact that no one who previously had C-19 caught it again during major outbreaks on a fishing vessel and at an overnight camp.
Time will tell more about the mutations of the virus that causes C-19, but current indications are that it will not dramatically mutate and become an ongoing cause of numerous deaths.
Years Of Lost Life
Beyond raw numbers of deaths, another crucial factor in measuring the deadliness of a public health threat is the years of life that it robs from its victims. In the words of the CDC, “the allocation of health resources must consider not only the number of deaths by cause but also by age.” Hence, the “years of potential life lost” has “become a mainstay in the evaluation of the impact of injuries on public health.”
This doesn’t mean that younger people are more important than older people, but it recognizes and accounts for the facts that:
- humans cannot ultimately prevent death; they can only delay it.
- there is a material difference between a malady that kills a 20 year-old in the prime of her life and one that kills a 90-year-old who would have otherwise died a month later.
These realities seem to be lost on some prominent medical authorities, like Howard Bauchner, the editor-in-chief of the Journal of the American Medical Association. He coauthored an editorial comparing 100,000 deaths from C-19 “to the number of U.S. soldiers killed in World War I or in the Korean and Vietnam wars combined.” This is deceptive because it fails to account for the facts that the average:
- The lifespan of U.S. residents is 79 years.
- The age of U.S. residents who have died of COVID-19 is about 75 years.
- The age of U.S. soldiers killed in Vietnam was 23 years.
While ignoring those facts, Bauchner says that “death seems like it should be an accurate measure of the pandemic’s evolution and effects – the worst outcome, an unequivocal outcome.” Given that death is ultimately everyone’s outcome, it is a flawed measure of the pandemic’s severity because it is incomplete.
A much more complete measure of C-19’s lethality also includes the years of life lost by each victim. Yet, determining this is not a simple matter of subtracting their ages at the time of their deaths from the average U.S. lifespan. This is because:
- The average U.S. lifespan includes the deaths of young people, which drives this figure down relative to what it is for older people. So even though the average life expectancy from birth is 79 years, people who are 79 years old live an average of 10 more years.
- C-19 is more deadly to people with preexisting health problems, and these people have lower average life expectancies than healthy people of the same ages.
Based on the average life expectancies of people of different ages and the ages of people who have died from C-19, Just Facts calculates that C-19 robs an average of 13.5 years from each of its victims. This is an overestimate because it does not account for preexisting health conditions, and one working paper calculates that the actual figure could be as low as 1.76 years in the United States.
Even based on the exaggerated figure of 13.5 years, the average years of life for each person who dies of C-19 is much less than other common causes of untimely death. For example, accidents rob an average of 30.6 years of life from each victim, while suicides cost an average of 35.6 years. And once again, these deaths occur every year, while C-19 is unlikely to have an ongoing high death toll because of its vulnerability to antibodies and limited prospects for mutation.
At a March 30 White House press conference, Dr. Deborah Birx, a world-renowned immunologist, presented a slide of projections for C-19 fatalities based on “five or six international and domestic modelers from Harvard, from Columbia, from Northeastern, from Imperial who helped us tremendously.” The model projected that 100,000 to 240,000 deaths would occur if Americans followed social distancing and hygiene guidelines. Birx added that “we really believe and hope every day that we can do a lot better than that because that’s not assuming 100% of every American does everything that they’re supposed to be doing, but I think that’s possible.”
The high-end of that range has already come to pass, but even if 500,000 U.S. residents eventually die from C-19 at an inflated average of 13.5 years of lost life per person, the disease will rob 6.8 million years of life from all Americans who were alive at the outset of 2020. In comparison, the flu will rob them of about 35 million years, suicides will rob them of 132 million years, and accidents will rob them of 409 million years.
Those figures reveal that accidents are about 60 times more lethal to Americans than this nightmare scenario for C-19. Likewise, the flu is about five times as lethal, and suicides are about 20 times as lethal. This is a substantially more comprehensive measure of deadliness than the tally of lives lost during a year – or any other random unit of time – because it accounts for the entirety of people’s lives and the total years of life that they lose.
On March 3, the World Health Organization announced that “about 3.4% of reported COVID-19 cases have died,” and “by comparison, seasonal flu generally kills far fewer than 1% of those infected.” Media outlets widely reported these figures and lambasted President Donald Trump for saying, “I think the 3.4% is really a false number” and “I would say the number is way under 1%.”
On March 11, Dr. Anthony Fauci, director of the National Institute of Allergy and Infectious Diseases, told a U.S. congressional committee that the mortality rate of C-19 is “somewhere around 1%, which means it is 10 times more lethal than the seasonal flu.”
Contrary to the statements of the World Health Organization and Fauci, Oxford University’s Center for Evidence-Based Medicine currently estimates that the fatality rate for C-19 is “somewhere between 0.1% and 0.35%.” This is well below 1%, just as Trump stated, and it nowhere near 10 times the flu fatality rate, which has averaged about 0.15% over the past nine years in the United States.
Similarly, the CDC’s current best estimates for the C-19 fatality rate are:
- 0.003% for people aged 0–19 years.
- 0.02% for people aged 20–49 years.
- 0.5% for people aged 50–69 years.
- 5.4% for people aged 70+ years.
In August, Cambridge University Press published a paper analyzing why early coverage of the C-19 fatality rate was so inaccurate. The study found that purveyors of inflated mortality rates likely confused two different measures of mortality called the “case fatality rate” and the “infection fatality rate.” The first of these is rate of death among confirmed cases of the disease, while the second is the rate among everyone who had it, including those who were asymptomatic or never diagnosed with it. The paper notes that misinformation about the death rate “helped launch a campaign of social distancing, organizational and business lockdowns, and shelter-in-place orders.”
At the time that those exaggerated mortality rates were being broadcast, ample evidence had already undercut them. This is proven by the remainder of this section, which is word-for-word from an article published by Just Facts on March 17:
Initial media reports of a 2–3% mortality rate for COVID-19 are inflated, and the actual figure may be closer to that of the flu, which has averaged about 0.15% over the past nine years in the United States. A large degree of uncertainty surrounds this issue due to the same factor that prevents accurate counts of infections: unreported cases.
As explained by Dr. Brett Giroir – who has authored nearly 100 peer-reviewed scientific publications and serves as the assistant secretary for health at the U.S. Department of Health and Human Services – the COVID-19 death rate is “lower than you heard probably in many reports” because the bulk of people who contract coronavirus don’t get seriously ill, and thus, many of them never get tested.
Giroir calls this a “denominator problem” because if you’re “not very ill, as most people are not, they do not get tested. They do not get counted in the denominator.” Giroir’s best estimate is that the mortality rate is probably “somewhere between 0.1% and 1%.” This ‘is likely more severe in its mortality rate than the typical flu’ rate of 0.1% to 0.15%, “but it’s certainly within the range.”
Giroir’s estimate accords with a February commentary in the New England Journal of Medicine by Fauci and others:
If one assumes that the number of asymptomatic or minimally symptomatic cases is several times as high as the number of reported cases, the case fatality rate may be considerably less than 1%. This suggests that the overall clinical consequences of COVID-19 may ultimately be more akin to those of a severe seasonal influenza (which has a case fatality rate of approximately 0.1%) or a pandemic influenza (similar to those in 1957 and 1968) rather than a disease similar to SARS or MERS, which have had case fatality rates of 9 to 10% and 36%, respectively.
A prime example of how journalists misreport on this issue is a March 12 article in Business Insider by Andy Kiersz. In this piece, he compares the “death rates” of COVID-19 from the South Korean CDC to that of the flu from the United States CDC. Based on these numbers, he reports that “South Korea – which has reported some of the lowest coronavirus death rates of any country – still has a COVID-19 death rate more than eight times higher than that of the flu.”
What Kiersz and his editors fail to understand is that the denominator for the Korean rate is the number of “confirmed cases,” while the denominator for the U.S. rate is based on a “mathematical model.” The CDC clarifies how the model works by citing a study on swine flu, which multiplies “43,677 laboratory-confirmed cases” of the disease by 41 to 131 times to calculate the denominator for the death rate. In the authors’ words, they do this because confirmed cases are:
likely a substantial underestimate of the true number. Correcting for under-ascertainment using a multiplier model, we estimate that 1.8 million–5.7 million cases occurred, including 9,000–21,000 hospitalizations.
Put simply, COVID-19 death rates that are based upon reported or confirmed infections grossly undercount the number of people with the disease. This, in turn, makes the death rate seem substantially higher than reality.
Another important factor in weighing the risks posed by COVID-19 is its transmissibility, or how contagious it is. In this respect, COVID-19 is much more dangerous than the seasonal flu because it spreads very quickly and can overwhelm hospitals.
Scientists measure contagiousness with a basic reproduction number, which is the average number of people who tend to catch a disease from each person who has it. This measure is an innate characteristic of the disease because it doesn’t account for actions that people take to prevent it. A February paper in the Journal of Travel Medicine explains that any disease with a basic reproduction number above 1.0 is likely to multiply over time.
The same paper evaluates 12 studies of the basic reproduction number of COVID-19 in various nations and finds that they “ranged from 1.4 to 6.49,” with an average of 3.28 and a median of 2.79. Based on their analysis of these studies, the authors conclude that the basic reproduction number of C-19 will likely prove to be “around 2–3” after “more data are accumulated.”
In comparison to the flu, a 2014 paper in the journal BMC Infectious Diseases analyzes 24 studies of the seasonal flu and finds that the median result for the basic reproduction number is 1.28. The authors stress that the seemingly small difference between 1.28 and higher figures like 1.80 “represent the difference between epidemics that are controllable and cause moderate illness and those causing a significant number of illnesses and requiring intensive mitigation strategies to control.”
All of the facts above were published in scientific journals by February – but in direct contradiction to them – the director-general of the World Health Organization gave a press conference on March 3 in which he stated that “COVID-19 spreads less efficiently than flu.”
A range of media outlets echoed that dangerous falsehood, as they did with others from the World Health Organization about immunity and fatality rates. Then with no apparent regard for the World Health Organization’s track record of spurious claims, Google-YouTube adopted a policy in May of censoring people who post content “that contradicts the World Health Organization (WHO) or local health authorities’ medical information about COVID-19.”
The high transmissibility of COVID-19 means that hygienic practices, social distancing, and other precautionary measures can save more lives than doing the same for common diseases like the flu.
There are, however, mortal dangers in overreacting because measures to limit the spread of C-19 have tangible impacts that can cost multitudes of lives. Numerous facts have shown that lockdowns, panic, and other responses to the pandemic have done just that. A small sampling of these include the following:
- The authors of a July paper in the Journal of the American Medical Association found:
- There is “an alarming trend across the U.S., where community members experiencing a health emergency are staying home – a decision that can have long-term, and sometimes fatal, consequences.”
- In the five states that had the most COVID-19 deaths in March and April, heart disease deaths were 89% above normal, and diabetes deaths were 96% above normal.
- “New York City’s death rates alone rose a staggering 398% from heart disease and 356% from diabetes.”
- A scientific survey commissioned by the American College of Emergency Physicians in April found that 29% of adults have “actively delayed or avoided seeking medical care due to concerns about contracting” C-19.
- A California-based ABC News station reported in May:
- “Doctors at John Muir Medical Center in Walnut Creek say they have seen more deaths by suicide during this quarantine period than deaths from the COVID-19 virus.”
- Mike deBoisblanc, head of the trauma unit at the hospital stated that he’s “seen a year’s worth of suicide attempts in the last four weeks,” and “mental health is suffering so much” that he says “it is time to end the shelter-in-place order.”
- A scientific survey conducted by the CDC in July found that about 32% of U.S. adults had “symptoms of anxiety disorder” as compared to 8% around the same time last year. The perils of this are underscored by a 2015 meta-analysis in the Journal of the American Medical Association Psychiatry, which found that the overall risk of death among people with anxiety is 43% higher than the general population.
- A study published by the American Medical Association in September found that 27.8% of U.S. adults had symptoms of depression during the C-19 pandemic as compared to 8.5% before the pandemic. The same 2015 meta-analysis found that depression is associated with a 71% higher risk of death.
- An article published by the Federal Reserve Bank of San Francisco estimated that “more than 20 million jobs” were “swept away” in the early months of the C-19 pandemic. The potential repercussions of this are highlighted by a 2011 meta-analysis in the journal Social Science & Medicine about mortality, “psychosocial stress,” and job losses. It found that “unemployment is associated with a 63% higher risk of mortality in studies controlling for covariates.”
- A scientific survey conducted by the Kaiser Family Foundation in late April found that 55% of U.S. workers “have lost their jobs or had a reduction in hours or pay as a result of” reactions to C-19. Regardless of whether these losses are brief or sustained, the same 2011 meta-analysis found that the correlation between unemployment and death “is significant in both the short and long term,” lending “some support to the hypothesis and previous findings that both the stress and the negative lifestyle effects associated with the onset of unemployment tend to persist even after a person has regained a job.”
- Four federal bills passed to address the pandemic and buffer the economic fallouts of state-imposed shutdowns cost about $2.5 trillion, or an average of $19,000 for every household in the nation. This has played a key role in driving the national debt to the highest portion of the U.S. economy in the nation’s history. A broad range of facts show that this can have serious negative consequences, including lower wages, weak economic growth, increased inflation, higher taxes, reduced government benefits, or combinations of such results. These, in turn, impair people’s quality of life and can reduce their life expectancy.
- A study published by the journal European Psychiatry in May found that:
- governments failed to model the “increased short-term and long-term mortality” caused by the mental health impacts of COVID-19 mitigation strategies like social distancing and lockdowns.
- these actions increased “suicide, depression, alcohol use disorder, childhood trauma due to domestic violence, changes in marital status, and social isolation.”
- the average person in Switzerland will lose an average of about 0.205 years of life due to these mitigation measures.
- “this loss would be entirely borne by 2.1% of the population, who will suffer an average of 9.79” years of life lost.
- these figures are “likely to underestimate the true impact of the mitigation strategies,” and more life may be lost due to “economic adversity, changes to activities of daily living such as eating, sleeping, smoking and ordinary alcohol consumption, or decrease in medical provision to those who have health problems unrelated to COVID-19.”
- A study published by Just Facts in May found that anxiety related to C-19 will ultimately destroy more years of life than can possibly be saved by lockdowns. With regard to this study, the accomplished psychiatrist Joseph P. Damore, Jr. wrote that it “thoroughly answers the question about the cure being worse than the disease.”
These facts prove that overreacting to C-19 can ultimately kill more people than are saved. Calling attention to this point, a study published by the journal Disaster Medicine and Public Health Preparedness in June found:
Exaggerated levels of fear were driven by sensationalist media coverage during the COVID-19 pandemic. And yet, while the public was ordered to lockdown, overall costs and benefits to society from severe mitigation measures had not been assessed.
The implications of overreacting to COVID-19 or any other potential hazard are aptly summarized in a teaching guide published by the American Society for Microbiology. This book explains why “the factors driving your concept of risk – emotion or fact – may or may not seem particularly important to you,” but they certainly are because “there are risks in misperceiving risks.”
A Look Back & The Path Forward
Aggressive social distancing can extend the timeframe over which COVID-19 patients are infected and hospitalized, but it cannot by itself reduce those outcomes in the long run. This is because COVID-19 is so contagious that another outbreak will begin and quickly proliferate as soon as the distancing measures cease.
A slew of lethal outcomes over the ensuing months have proved that statement true, especially the global number of C-19 deaths per day:
A sample of articles from the New York Times shows that this post-lockdown carnage occurred across the globe and took many people by surprise:
- July 24: “As the pandemic continues to grow around the world … troubling resurgences have hit several places that were seen as models of how to respond to the virus, such as Hong Kong, Spain, and Australia.”
- July 29: “After months without a single coronavirus death, or even a confirmed case of local transmission, a new outbreak has struck Vietnam. And it’s spreading.”
- Oct. 17: “When the coronavirus began sweeping around the globe this spring, people from Seattle to Rome to London canceled weddings and vacations, cut off visits with grandparents and hunkered down in their homes for what they thought would be a brief but essential period of isolation. But summer did not extinguish the virus. And with fall has come another dangerous, uncontrolled surge of infections that in parts of the world is the worst of the pandemic so far.”
- Oct. 28: “But with countries like France, Italy and Britain reporting their highest death toll in months, and Germany facing soaring cases this week, the authorities are now scrambling to reimpose drastic measures to curb a surge in infections and hospitalizations that could result in more deaths than during the first wave of the pandemic.”
- Dec. 4: “We now know that the second wave in Europe has become deadlier than the first.”
All of this was predicted and predictable. Even the Imperial College of London, which provided the impetus for mass lockdowns, admitted in March that the “more successful a strategy is at temporary suppression, the larger the later epidemic is predicted to be in the absence of vaccination, due to lesser build-up of herd immunity.”
Making a similar point eight years before the pandemic, a 2012 paper in the journal PLoS One about “Immunity in Society” noted that:
when a sufficiently high proportion of individuals within a population becomes immune (either through prior exposure or through mass vaccination), community or ‘herd’ immunity emerges, whereby individuals that are poorly immunized are protected by the collective ‘immune firewall’ provided by immunized neighbors. In humans and other vertebrate communities … responses to a previously encountered pathogen are faster and stronger than those to a novel pathogen, and thus individuals are better at blocking its spread. [Emphasis added.]
Equally, if very few people are immune to a disease, they can transmit it to others instead of blocking it. Without a vaccine, the only way people can become immune to C-19 is by catching it and recovering. This means that too much social distancing may cause more deaths because young, healthy people – who would otherwise catch the disease, recover quickly, and become firewalls– remain as potential carriers.
Quantifying those dynamics, a study published by the British Medical Journal in October found these stunning and “counterintuitive results” about lockdowns in the United Kingdom:
- The advice given to United Kingdom policymakers was focused “on reducing total number of cases and not the number of deaths.”
- “Contrary to popular perception,” the Imperial College paper that was used to justify the lockdown never “specifically modelled” the lockdown.
- When adjusted to reflect high transmissibility of C-19, the Imperial College model projects that once some basic preventative measures are in place like quarantining infected people and social distancing for the elderly:
- the “closure of schools and universities would increase the total number of deaths.”
- “general social distancing” of the entire population “was also projected to increase the total number of deaths.”
- “postponing the spread of COVID-19 means that more people are still infectious and are available to infect older age groups, of whom a much larger fraction then die.”
Those outcomes assume a C-19 mortality rate of 1% (higher than reality) and that widespread vaccination does not occur for 800 days (probably slower than reality), but the basic point remains the same: Lockdowns to control the spread of C-19 can actually cause more C-19 deaths by preventing the development of herd immunity among healthy people – the vast majority of whom are very resilient to the virus.
And that only accounts for added C-19 deaths. In addition, lockdowns trigger hordes of other types of deaths both immediately and in the long-term by causing anxiety, depression, missed medical care, unemployment, and poverty.
Despite those facts, media outlets have often scorned the idea of herd immunity, like this October article in the New York Times that claims:
Many experts say ‘herd immunity’ – the point at which a disease stops spreading because nearly everyone in a population has contracted it – is still very far off. Leading experts have concluded, using different scientific methods, that about 85 to 90% of the American population is still susceptible to the coronavirus.
That statement suffers from two fatal flaws:
- It is not true that “nearly everyone in a population” must catch C-19 to achieve herd immunity. Even simplistic models – which assume that immunity is evenly distributed and that all age groups take the same precautions – estimate that herd immunity for C-19 emerges when about 50–67% of the population is immune. Using more realistic scenarios, an August paper in the journal Science places this figure at roughly 43%.
- The claim that 85–90% of Americans are still susceptible to C-19 is not based on substantially “different scientific methods” but only on studies that only test for antibodies, which can greatly understate immunity. As documented by the authors of a study conducted in Sweden and published by the journal Cell in August:
- “Our results indicate that public immunity to COVID-19 is probably significantly higher than antibody tests have suggested.”
- Most studies of C-19 immunity merely test for antibodies, but “antibody responses are not detectable in all patients, especially those with less severe forms of COVID-19,” and this is the case with “most people” who catch C-19.
- T cells – which are the body’s main source of lasting immunity – are present in the vast majority of people who have had C-19, and these cells respond like “those observed in the context of successful vaccines, suggesting that natural exposure or infection may prevent recurrent episodes of severe COVID-19” in people who don’t have antibodies.
- Nearly “twice as many exposed family members and healthy individuals who donated blood during the pandemic generated memory T cell responses versus antibody responses, implying that” testing for antibodies “has underestimated the extent of population-level immunity” for C-19.
Collectively, these data indicate that herd immunity could have been achieved with much less loss of life than has already occurred. That would have required doing more to protect vulnerable people and allowing young healthy people greater freedom instead of locking them down. This is precisely the opposite of what certain high-profile media figures called for and government officials have done.
For the purpose of illustration, 43% of the U.S. population – or roughly enough to achieve herd immunity – is under the age of 33. If everyone in this age group went about their business and caught the disease while everyone else stayed COVID-free through diligent social distancing, hygiene, testing, and other creative measures:
- 14,218 people under the age of 33 would have died based on the CDC’s best estimates for C-19 fatality rates at these ages.
- the people who died would have lost a total of 800,000 years of life.
In comparison, 240,213 or about 15 times more fatalities from C-19 were recorded among people of all ages in death certificates filed by Nov. 21. Given the ages of the deceased, this amounts to 3.2 million years of life, or four times worse than the idealized scenario.
That scenario unrealistically assumes prevention measures and herd immunity are so effective that no one over the age of 32 catches C-19 – but like the British Medical Journal paper – it shows that there comes a tipping point where broad lockdowns cause more C-19 deaths than balanced measures. Again, that’s in addition to all of the other deadly effects of lockdowns.
However, strategic social distancing can keep hospitalizations at reasonable levels so that victims receive proper care, and it can also buy time to discover and mass-produce effective treatments.
During a March 14 press conference, U.S. Surgeon General Jerome Adams asserted that “this situation will last longer, and more people will be hurt” if “we are complacent, selfish, uninformed,” and if “we spread fear, distrust, and misinformation.” Yet, that is exactly what many media outlets, governments, and big tech companies have done.
Conversely, Adams said that “we will overcome this situation” if we “pitch in” and “share the facts.” The essential facts above confirm the wisdom of his words.